Birth Defects Research Part A: Clinical and Molecular Teratology 
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Birth Defects Research Part A: Clinical and Molecular Teratology,Volume 106, Issue 12, Page 1042-1042, December 2016. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - October 12, 2016 Category: Perinatology & Neonatology Source Type: research
Letter to the Editor
(Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - October 11, 2016 Category: Perinatology & Neonatology Authors: Russell S. Kirby Tags: Correspondence Source Type: research
Letter to the editor interpreting trends in the context of previous evidence
(Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - October 11, 2016 Category: Perinatology & Neonatology Authors: Russell S. Kirby Tags: Letter to the Editor Source Type: research
Diprosopus: Systematic review and report of two cases
Birth Defects Research Part A: Clinical and Molecular Teratology,Volume 106, Issue 12, Page 993-1007, December 2016. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - October 5, 2016 Category: Perinatology & Neonatology Authors: Mar ía Paz Bidondo,
Boris Groisman,
Agostina Tardivo,
Fabián Tomasoni,
Verónica Tejeiro,
Inés Camacho,
Mariana Vilas,
Rosa Liascovich,
Pablo Barbero Source Type: research
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Birth Defects Research Part A: Clinical and Molecular Teratology,Volume 106, Issue 12, Page 993-1007, December 2016. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - October 5, 2016 Category: Perinatology & Neonatology Source Type: research
Prevention of neural tube defects in Lrp mutant mouse embryos by folic acid supplementation
ConclusionLrp2 is required for folate homeostasis in heterozygous dams and high levels of supplementation prevents NTDs. Furthermore, high levels of dietary iron supplementation interfered with folic acid supplementation negating the positive effects of supplementation in this model. Birth Defects Research (Part A), 2016. © 2016 The Authors Birth Defects Research Part A: Clinical and Molecular Teratology Published by Wiley Periodicals, Inc. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 30, 2016 Category: Perinatology & Neonatology Authors: Julia A. Sabatino, Bethany A. Stokes, Irene E. Zohn Tags: Original Research Article Source Type: research
Prevalence of neural tube defects in a rural area of north india from 2001 to 2014: A population ‐based survey
ConclusionWe found high prevalence of NTDs in rural community settings from north India for years 2001 to 2014.Birth Defects Research (Part A), 2016.© 2016 Wiley Periodicals, Inc. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 30, 2016 Category: Perinatology & Neonatology Authors: Shashi Kant, Sumit Malhotra, Arvind Kumar Singh, Partha Haldar, Ravneet Kaur, Puneet Misra, Neerja Gupta Tags: Original Research Article Source Type: research
Metabolite profiling of whole murine embryos reveals metabolic perturbations associated with maternal valproate ‐induced neural tube closure defects
ConclusionMaternal VPA treatment markedly perturbed purine and pyrimidine metabolism in E9.5 embryos. In combination with a high FA diet, VPA treatment resulted in gross metabolic changes, likely caused by a multiplicity of mechanisms, including an apparent disruption of mitochondrial beta‐oxidation. Birth Defects Research (Part A), 2016. © 2016 Wiley Periodicals, Inc. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 30, 2016 Category: Perinatology & Neonatology Authors: Darya Akimova, Bogdan J. Wlodarczyk, Ying Lin, M. Elizabeth Ross, Richard H. Finnell, Qiuying Chen, Steven S. Gross Tags: Original Research Article Source Type: research
Preaxial polydactyly following early gestational exposure to the smoothened agonist, SAG, in C57BL/6J mice
ConclusionPreaxial polydactyly can be caused in the developing embryo by acute maternal administration of a Smo agonist that activates HH signaling. These results are consistent with the preaxial polydactyly induced in developmental disorders associated with mutations in HH signaling genes.Birth Defects Research (Part A), 2016.© 2016 Wiley Periodicals, Inc. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 30, 2016 Category: Perinatology & Neonatology Authors: Eric W. Fish, Scott E. Parnell, Kathleen K. Sulik, Lorinda K. Baker, Laura B. Murdaugh, David Lamson, Kevin P. Williams Tags: Brief Report Source Type: research
Genetic backgrounds and modifier genes of NTD mouse models: An opportunity for greater understanding of the multifactorial etiology of neural tube defects
Neurulation, the early embryonic process of forming the presumptive brain and spinal cord, is highly complex and involves hundreds of genes in multiple genetic pathways. Mice have long served as a genetic model for studying human neurulation, and the resulting neural tube defects (NTDs) that arise when neurulation is disrupted. Because mice appear to show mostly single gene inheritance for NTDs and humans show multifactorial inheritance, mice sometimes have been characterized as a simpler model for the identification and study of NTD genes. But are they a simple model? When viewed on different genetic backgrounds, many gen...
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 30, 2016 Category: Perinatology & Neonatology Authors: Renee Y.M. Leduc, Parmveer Singh, Heather E. McDermid Tags: Review Article Source Type: research
Acardiac twin pregnancies part III: Model simulations
Birth Defects Research Part A: Clinical and Molecular Teratology,Volume 106, Issue 12, Page 1008-1015, December 2016. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 15, 2016 Category: Perinatology & Neonatology Authors: Martin J.C. Gemert,
Michael G. Ross,
Peter G.J. Nikkels,
Jeroen P.H.M. van den Wijngaard Source Type: research
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Birth Defects Research Part A: Clinical and Molecular Teratology,Volume 106, Issue 12, Page 1008-1015, December 2016. (Source: Birth Defects Research Part A: Clinical and Molecular Teratology)
Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 15, 2016 Category: Perinatology & Neonatology Source Type: research
