Clinicopathologic features and outcomes of primary cardiac tumors: a 16-year-experience with 212 patients at a Chinese medical center
The objective of this study was to review the clinicopathologic features and surgical outcomes of patients with primary cardiac tumors in a single medical center in China. We have retrospectively reviewed 212 consecutive adult patients who underwent surgical resection of primary cardiac tumors at our center from January of 2000 to June of 2017. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - January 5, 2018 Category: Cardiology Authors: Ji-Gang Wang, Baolei Wang, Yanjiao Hu, Ji-Hua Liu, Bing Liu, Hui Liu, Peng Zhao, Lijuan Zhang, Yu-Jun Li Tags: Original Article Source Type: research
Surgical pathology of subaortic septal myectomy: histology skips over clinical diagnosis
Subaortic septal myectomy is usually performed to mitigate obstruction in patients with the obstructive form of hypertrophic cardiomyopathy (HCM) or in those with congenital subaortic stenosis. Moreover, it is combined with aortic valve replacement in patients with severe aortic valve stenosis (SAS) and asymmetrical septal hypertrophy causing concomitant left ventricular outflow tract obstruction. When both conditions coexist, it is conceptually difficult to identify a cardiomyopathy beyond an adaptive myocardial hypertrophy, strictly related to pressure overload. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - January 2, 2018 Category: Cardiology Authors: Jo ão Abecasis, Rosa Gouveia, Mariana Castro, Maria João Andrade, Regina Ribeiras, Sância Ramos, Miguel Abecasis, Nuno Cardim, Victor Gil Tags: Original Article Source Type: research
Surgical pathology of subaortic septal myectomy: hystology skips over clinical diagnosis
subaortic septal myectomy is usually performed to mitigate obstruction in patients with the obstructive form of hypertrophic cardiomyopathy (HCM) or in those with congenital subaortic stenosis. Moreover, it is combined with aortic valve replacement in patients with severe aortic valve stenosis (SAS) and asymmetrical septal hypertrophy causing concomitant left ventricular outflow tract obstruction. When both conditions coexist it is conceptually difficult to identify a cardiomyopathy beyond an adaptive myocardial hypertrophy, strictly related to pressure overload. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - January 2, 2018 Category: Cardiology Authors: Jo ão Abecasis, Rosa Gouveia, Mariana Castro, Maria João Andrade, Regina Ribeiras, Sância Ramos, Miguel Abecasis, Nuno Cardim, Victor Gil Source Type: research
Survival kinase-dependent pathways contribute to gender difference in the response to myocardial ischemia –reperfusion and ischemic post-conditioning
The response to ischemia/reperfusion and the effects of ischemic post-conditioning (IPC) are sex-dependent, but the mechanisms have not been clarified. Male (M) and female (F) rat hearts isolated and perfused using the Langendorff technique were subject to 30 min of global ischemia (GI) and 60 min reperfusion (R). In IPC hearts, three cycles of 30-sec GI/30-sec R were applied at the beginning of R. Infarct size and myocardial function were assessed. Superoxide production, antioxidant systems, and expressions of phosphorylated forms of serine/threonine kinase (Akt), glycogen synthase kinase 3 β (GSK-3β), protein kinase C ...
Source: Cardiovascular Pathology - December 28, 2017 Category: Cardiology Authors: Alejandro Ciocci Pardo, Sergio Scuri, Luisa F. Gonz ález Arbeláez, Claudia Caldiz, Juliana Fantinelli, Susana M. Mosca Tags: Original Article Source Type: research
Characteristics, survival and incidence rates and trends of primary cardiac malignancies in the United States
The available literature on the incidence, management and prognosis of primary malignant cardiac tumors [PMCTs] is limited to single-center studies, prone to small sample size and referral bias. We used data from the Surveillance, Epidemiology, and End Results [SEER]-18 registry (between 2000 and 2014) to investigate the distribution, incidence trends and the survival rates of PMCTs. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - December 28, 2017 Category: Cardiology Authors: Anas M. Saad, Abdelrahman Ibrahim Abushouk, Muneer J. Al-Husseini, Sami Salahia, Anas Alrefai, Ahmed M. Afifi, Mohamed M. Abdel-Daim Tags: Original Article Source Type: research
Survival kinase-dependent pathways contribute to gender difference in the response to myocardial ischemia –reperfusion and ischemic post-conditioning
The response to ischemia/reperfusion and the effects of ischemic post-conditioning (IPC) are sex-dependent, but the mechanisms have not been clarified. Male (M) and female (F) rat hearts isolated and perfused using the Langendorff technique were subject to 30 min of global ischemia (GI) and 60 min reperfusion (R). In IPC hearts, three cycles of 30-sec GI/30-sec R were applied at the beginning of R. Infarct size and myocardial function were assessed. Superoxide production, antioxidant systems, and expressions of phosphorylated forms of serine/threonine kinase (Akt), glycogen synthase kinase 3 β (GSK-3β), protein kinase C ...
Source: Cardiovascular Pathology - December 28, 2017 Category: Cardiology Authors: Alejandro Ciocci Pardo, Sergio Scuri, Luisa F. Gonz ález Arbeláez, Claudia Caldiz, Juliana Fantinelli, Susana M. Mosca Tags: Original Article Source Type: research
Survival Kinases-Dependent Pathways Contribute to Gender Difference in the Response to Myocardial Ischemia-Reperfusion and Ischemic Post-Conditioning
The response to ischemia/reperfusion and the effects of ischemic post-conditioning (IPC) are sex-dependent, but the mechanisms have not been clarified. Male (M) and female (F) rat hearts isolated and perfused using the Langendorff technique were subject to 30 min of global ischemia (GI) and 60 min reperfusion (R). In IPC hearts, three cycles of 30-sec GI/30-sec R were applied at the beginning of R. Infarct size and myocardial function were assessed. Superoxide production, antioxidant systems, and expressions of phosphorylated forms of serine/threonine kinase (Akt), glycogen synthase kinase 3 β (GSK-3β), protein kinase C ...
Source: Cardiovascular Pathology - December 28, 2017 Category: Cardiology Authors: Alejandro Ciocci Pardo, Sergio Scuri, Luisa F. Gonz ález Arbeláez, Claudia Caldiz, Juliana Fantinelli, Susana M Mosca Source Type: research
Characteristics, Survival and Incidence Rates and Trends of Primary Cardiac Malignancies in the United States
The available literature on the incidence, management and prognosis of primary malignant cardiac tumors [PMCTs] is limited to single-center studies, prone to small sample size and referral bias. We used data from the Surveillance, Epidemiology, and End Results [SEER]-18 registry (between 2000 and 2014) to investigate the distribution, incidence trends and the survival rates of PMCTs. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - December 28, 2017 Category: Cardiology Authors: Anas M. Saad, Abdelrahman Ibrahim Abushouk, Muneer J. Al-Husseini, Sami Salahia, Anas Alrefai, Ahmed M Afifi, Mohamed M. Abdel-Daim Source Type: research
Editorial Board
(Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - December 12, 2017 Category: Cardiology Source Type: research
Table of Contents
(Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - December 12, 2017 Category: Cardiology Source Type: research
Rare case of infective endocarditis involving mitral annular calcification leading to hemopericardium and sudden cardiac death: a case report
We present a unique case of a 76-year-old female who had sudden cardiac death due to hemopericardium. Post mortem examination revealed infective endocarditis(IE) superimposing on mitral annulus calcification(MAC) leading to abscess formation, epicardial fistulation and hemopericardium. MAC is currently considered a relatively benign condition of the elderly [1], however evidence suggests there are severe consequences when these patients develop IE. With an aging population, this is likely to be a more frequent occurrence and should be considered in patients who present with bacteremia of unknown origin. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - December 4, 2017 Category: Cardiology Authors: Shauna Wentzell, Vidhya Nair Tags: Clinical Case Report Source Type: research
Rare case of infective endocarditis involving mitral annular calcification leading to hemopericardium and sudden cardiac death: A case report
We present a unique case of a 76-year-old female who had sudden cardiac death due to hemopericardium. Post mortem examination revealed infective endocarditis(IE) superimposing on mitral annulus calcification(MAC) leading to abscess formation, epicardial fistulation and hemopericardium. MAC is currently considered a relatively benign condition of the elderly [1], however evidence suggests there are severe consequences when these patients develop IE. With an aging population, this is likely to be a more frequent occurrence and should be considered in patients who present with bacteremia of unknown origin. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - December 4, 2017 Category: Cardiology Authors: Shauna Wentzell, Vidhya Nair Tags: Case Report Source Type: research
TUG1 knockdown ameliorates atherosclerosis via up-regulating the expression of miR-133a target gene FGF1
Long non-coding RNAs (lncRNAs) have been revealed to participate in the pathological events associated with atherosclerosis. However, the exact role of lncRNA taurine-up-regulated gene 1 (TUG1) and its possible molecular mechanism in atherosclerosis remain unidentified. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - November 26, 2017 Category: Cardiology Authors: Lei Zhang, Hailing Cheng, Yuxia Yue, Shuangzhan Li, Daping Zhang, Ruili He Tags: Original Article Source Type: research
TUG1 knockdown ameliorates atherosclerosis via up-regulating the expression of miR-133a target gene FGF1
Long non-coding RNAs (lncRNAs) have been revealed to participate in the pathological events associated with atherosclerosis. However, the exact role of lncRNA taurine-upregulated gene 1 (TUG1) and its possible molecular mechanism in atherosclerosis remain unidentified. (Source: Cardiovascular Pathology)
Source: Cardiovascular Pathology - November 26, 2017 Category: Cardiology Authors: Lei Zhang, Hailing Cheng, Yuxia Yue, Shuangzhan Li, Daping Zhang, Ruili He Source Type: research
