Kcnk3 dysfunction exaggerates the development of pulmonary hypertension induced by left ventricular pressure overload

ConclusionsOur results indicate thatKcnk3-LOF is a key event in the pathobiology of PH due to AAC, suggesting that Kcnk3 channel dysfunction could play a potential key role in the development of PH due to LHD.
Source: Cardiovascular Research - Category: Cardiology Source Type: research