Human CD16+ monocytes promote a pro-atherosclerotic endothelial cell phenotype via CX3CR1 –CX3CL1 interaction

ConclusionBy demonstrating endothelial proatherosclerotic gene regulation in direct contact with CD16+ monocytes, in part via cellular CX3CR1 –CX3CL1 interaction, our data delineate a mechanism how this celltype can increase cardiovascular risk.
Source: Cardiovascular Research - Category: Cardiology Source Type: research