LncRNA PRADX-mediated recruitment of PRC2/DDX5 complex suppresses UBXN1 expression and activates NF- κ B activity, promoting tumorigenesis

Conclusions: PRADX acts as a cancer driver and may serve as a potential therapeutic target for glioblastoma and colon adenocarcinoma.
Source: Theranostics - Category: Molecular Biology Authors: Tags: Research Paper Source Type: research