Ginsenoside compound-Mc1 attenuates oxidative stress and apoptosis in cardiomyocytes through an AMPK-dependent mechanism

ConclusionGinsenoside Mc1 decreases oxidative stress and increases cell viability in H9c2 cells and the heart tissue isolated from HFD-fed mice via an AMPK-dependent mechanism, suggesting its potential as a novel therapeutic agent for oxidative stress-related cardiac diseases.
Source: Journal of Ginseng Research - Category: Complementary Medicine Source Type: research