TGF ‐β1 promotes gap junctions formation in chondrocytes via Smad3/Smad4 signalling
ConclusionsFor the first time, these results revealed a vital role of TGF ‐β1 in cell‐cell communication in chondrocytes via gap junction formation. We describe the regulatory mechanism, the involvement of TGF‐β type I receptor and the nuclear translocation of Smad3/4.
Source: Cell Proliferation - Category: Cytology Authors: Qingxuan Wang,
Chenchen Zhou,
Xiaobing Li,
Linyi Cai,
Jing Zou,
Demao Zhang,
Jing Xie,
Wenli Lai Tags: ORIGINAL ARTICLE Source Type: research