Increased oxidative stress and CaMKII activity contribute to electro ‐mechanical defects in cardiomyocytes from a murine model of Huntington's disease

This article is protected by copyright. All rights reserved.

Source: FEBS Journal - November 19, 2018 Category: Research Authors: Julliane Vasconcelos Joviano ‐Santos, Artur Santos‐Miranda, Ana Flávia Machado Botelho, Itamar Couto Guedes de Jesus, Jéssica Neves Andrade, Tatiane de Oliveira Barreto, Matheus Proença S. Magalhães‐Gomes, Priscila Aparecida Costa Valadã Tags: Original Article Source Type: research