MiR ‐182 inhibits the epithelial to mesenchymal transition and metastasis of lung cancer cells by targeting the Met gene
Conclusions: miR‐182 might suppress the EMT and metastasis via inactivation of Met/AKT/Snail in non‐small cell lung cancer (NSCLC) cells, which implicates miR‐182 may be useful as a new therapeutic target in NSCLC. This article is protected by copyright. All rights reserved
Source: Molecular Carcinogenesis - Category: Molecular Biology Authors: Yongwen Li, Hongbin zhang, Ying Li, Chenlong Zhao, Yaguang Fan, Jinghao Liu, Xin Li, Hongyu Liu, Jun Chen Tags: RESEARCH ARTICLE Source Type: research
More News: Cancer | Cancer & Oncology | Epithelial Cancer | Genetics | Lung Cancer | Molecular Biology | Non-Small Cell Lung Cancer | Study