Endothelial damage effects of circulating microparticles from patients with stable angina is reduced by aspirin though ERK/p38 MAPKs pathways

ConclusionThese results indicate that the pro‐inflammatory, oxidative stress, procoagulant, and adhesion properties of MPs can be partly blocked by aspirin via the ERK‐NO/O2‐ and p38 ‐NF‐κB‐VCAM‐1signalpathway, which clarified other functions beyond anti‐atherothrombotic of aspirin.This article is protected by copyright. All rights reserved.

Source: Cardiovascular Therapeutics - May 18, 2017 Category: Cardiology Authors: Cheng Gong, Xue ‐Feng Shan, Xu‐Lang Wang, Wei‐Wei Dong, Zhe Li, D Xin‐Hong Liu, Wei Zhang, Kun Xing, Feng‐Jun Chang Tags: Original Research Article Source Type: research