Salidroside protects against homocysteine ‐induced injury in human umbilical vein endothelial cells via the regulation of endoplasmic reticulum stress

ConclusionsTaken together, these findings implicate that SAL could regulate ER stress pathway on the viability of endotheliocyte induced by Hcy in vitro. Our findings provide the first evidence that SAL plays an important role in endotheliocyte protection via suppressing ER stress pathway in HUVEC cells and that it may be a promising therapeutic target for atherosclerosis and cardiovascular disease.
Source: Cardiovascular Therapeutics - Category: Cardiology Authors: Tags: Original Research Article Source Type: research