Reduction in cadmium‐induced toxicity by c‐Jun modulation in mouse embryo limb bud cells

ConclusionThe JNK pathway, and particularly the downstream effector c‐Jun, appears to play an important role in regulating cell survival and differentiation in mouse embryo limb bud cells both in the presence and absence of the toxic metal cadmium. Birth Defects Research (Part A), 2015. © 2015 Wiley Periodicals, Inc.

Source: Birth Defects Research Part A: Clinical and Molecular Teratology - September 26, 2015 Category: Perinatology & Neonatology Authors: Carolyn M. Kapron, Le Cheng Tags: Original Research Source Type: research